Data on Diphtheria
Ekanem's earlier noted research (Table XI), reveals an increase of 215 percent in the number of diphtheria cases by the end of the three year period following implementation of UNICEF's Expanded Program of Immunization. Robert Mendelsohn (Assoc. Prof. of Preventive Medicine and Community Health, University of Illinois) reports "that children who have been immunized [for diphtheria] fare no better than those who have not."
He went on to describe an outbreak of diphtheria in which "fourteen of twenty-three carriers had been fully immunized." This means that just over 60 percent of the carriers who were presumed to be protected by the toxoid, contracted the disease. In his words
"Episodes such as these shatter the argument that immunization can be credited with eliminating diphtheria or any of the other . . . childhood diseases."73
The following conclusion is extracted from the Minutes of the 15th Session (November 20-21, 1975) of the Panel of Review of Bacterial Vaccines and Toxoids with Standards and Potency (data presented by the US Bureau of Biologics, and the Food and Drug Administration).
For several reasons, diphtheria toxoid, fluid or absorbed, is not as effective an immunizing agent as might be anticipated. Clinical (symptomatic) diphtheria may occur . . . in immunized individuals--even those whose immunization is reported as complete by recommended regimes . . . the permanence of immunity induced by the toxoid . . . is open to question.74
Earlier historical data on protective toxoiding efforts in N. America clearly verify not only the FDA's conclusion, but the fact that the toxoid actually exacerbated the seriousness of the disease. North American data on various diphtheria outbreaks in the early 40's, reveal the following facts.
- In the Halifax Canada epidemic, of the cases admitted for hospital treatment, 66 had previously received one or more doses of diphtheria toxoid or antitoxin, or were found Shick negative. In fact, of this number five cases had been immunized within the preceding two month period.75
- In the Ottawa Canada epidemic, of 99 cases (all under the age of 15), 36 were found to have previously received all three doses of the toxoid.76
- In the Baltimore USA epidemic, 63 percent of all cases had a record or history of prior immunization with toxoid. Among the fatal and more serious "Bull-neck" cases, 77.8 percent had previously been toxoided.77
- During roughly the same historic period, we find in various European countries a gripping picture suggesting that the use of Diphtheria toxoid in fact precipitated epidemics of the disease.77
- Throughout 1941 to 1944 "The Ministry and Dept. of Health, Scotland, admitted almost 23,000 cases of diphtheria in immunized children," with 180 fatalities.78
- By the year 1941, the majority of children in France had been inoculated for diphtheria, the case rate standing at 13,795 by the end of that year. Mass immunization efforts continued, and "by 1943, the diphtheria cases were more than tripled to 46,750."79
- Diphtheria increased by 55 percent in Hungary.
- Diphtheria tripled in Geneva, Switzerland after the introduction of compulsory immunization laws.
- In Germany, with compulsory mass immunization "introduced in 1940, the number of cases increased from 40,000 per year to 250,000 by 1945, virtually all among immunized children."
- Norway, during the same time frame--just noted--remained unvaccinated, and had only 50 recorded cases of diphtheria. 80
- "In Sweden, diphtheria virtually disappeared without any immunization."81
- According to Coumoyer's research, official US Military records show that enlisted men and women who are thoroughly vaccinated--manifest a morbidity and mortality rate from diphtheria four times higher, than that of unvaccinated civilians.82
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Data on Measles
As already noted earlier in this report, the national per capita case rate in Thailand for measles in 1982, 2 years before the advent of the Expanded Programme of Immunization, was lower than in the year 1988, i.e., 5 years after implementation of EPI. Per Ekanem's earlier cited research, the national per capita case rate in Nigeria for measles in 1973, 6 years before the advent of UNICEF's Expanded Programme of Immunization, was lower than in the year 1982, i.e., 3 years after implementation of EPI.83
The University of Alberta initiated special research on the question of measles immunity, as a result of a measles epidemic which "swept" the University campus in 1987, despite a "98 percent immunization rate." The research team's head immunologist R. Marusyk (who is also affiliated with the Alberta Provincial Public Health Laboratory) has subsequently confirmed that: it is an invalid assumption that vaccination programs for measles--which are normally administered at 9 to 12 months, and a later childhood booster shot--confers lifelong immunity.
One of their findings indicated that 93 percent of infants "who were studied" showed no immunity by the age of six months. The mothers of the 120 babies had all been vaccinated. Normally, antibodies that have been transferred at birth from the mother to the child remain present for a year."84
(According to D. de Saving at IDRC, this transfer and retention of antibodies apparently occurs when the mother has had an actual measles infection, and not just vaccination.)
Similar to the experience at the University of Alberta, the National Geographic in its January 1991 issue article "The Disease Detectives," refers to a 1988 measles epidemic at Fort Lewis College, Durango, Colorado USA in these words:"Surprisingly most who fell ill had been vaccinated. CDC (US Center for Disease Control) investigators rushed to the campus during the 1988 outbreak to trace what had gone wrong."
There are repeated reports of measles epidemics occurring in fully vaccinated populations. These failures have occurred repeatedly since the vaccines introduction. 85
Other documented research findings follow:
- A survey conducted in 1978--covering 30 states in the US--revealed that "more than half of the children who contracted measles had been adequately vaccinated;"86
- Moskowitz et al. found that in those states with comprehensive (k-grade 12) immunization requirements, between 61 and 90 percent of measles cases occur in persons who received the recommended vaccines;87 and
- A review of 1,600 cases of measles in Quebec, Canada in the period of January to May of 1989, revealed that 58 percent of school-age cases had been previously vaccinated.88
According to an unpublished WHO research study comparing what would be defined as a "measles susceptible" group of children, to a control group that had been immunized for measles, it was observed that: the non-immunized group manifested a normal contraction rate of 2.4 percent, whereas the immunized group exhibited a 33.5 percent contraction level.
This implies a 15 times greater likelihood of infection by the immunized.89 (The researchers responded to these results with the comment that the vaccine must have been mishandled, or perhaps the vaccine used was badly manufactured.)
It is of interest that there is an emerging body of mathematically based epidemiological research which suggests practicable problems with EPI efforts in the control and eradication of measles in the Developing World. For example, P. Kenya observes that:
Horizontal mass immunization campaigns at regular intervals may be impractical in terms of costs and operational logistics. . . . In spite of high measles immunization coverages, measles epidemics are often reported, not only in the less developed regions but also in those developed countries with measles elimination targets.90
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Data on Polio
An article in a major consumer journal titled "Twentieth-century miraclemaker," in extolling the value of Salk's polio vaccine, indicated that in 1953, there were 15,600 cases of paralytic polio in the United States; by 1957, due to the vaccine, this number dropped to 2,499." Since this popular conception persists to this day as an important demonstration of the effectiveness of vaccination procedures in general, and the polio vaccine in particular, it bears some re-examination.
Bernard Greenberg (late Dean--School of Public Health, University of N. Carolina) who--during the polio epidemics of the 50's--chaired the Committee on Evaluation and Standards for the American Public Health Association, submitted testimony to the Congressional Hearings on polio vaccines (HR0541, 1962).
His evidence respecting diagnostic modifications and statistical manipulation, seriously challenged the popularly promoted view that the epidemics subsided as a result of vaccine intervention. In his words "As a result of . . . changes in both diagnosis and diagnostic methods, the rates of paralytic poliomyelitis plummeted from the early 1950's to a low in 1957." This involved:
- redefinition of what constitutes an epidemic
- redefinition of the disease; and
- mislabelling, and later reclassification (prior to 1954 "large numbers" of presumed "paralytic polio" cases were actually "Coxsackie . . . and aseptic meningitis," statistical reclassification of "polio" cases (not leading to permanent paralysis) in the ensuing 4 year period became the norm in virtually all regions of the country.
It is of further interest that Greenberg testified that after the introduction of much more intensive and frequently compulsory immunization programs--beginning in 1957--there was a correspondingly substantial increase in polio cases (which were presumably paralytic, due to the aforenoted reclassification process).
In the period of 1957-1958 there was a 50 percent increase, and 1958-1959 an 80 percent increase in such cases. He also indicated that during this period statistics were manipulated and statements made by the US Public Health service, to give an opposite impression.92
A distinguished interdisciplinary medical panel moderated at the 120th Annual Meeting of the Illinois State Medical Society, confirmed that in the year 1959, roughly 1,000 cases of paralytic polio occurred in persons who had previously received multiple doses of the Salk vaccine. As a panel member,B. Greenberg contributed the following observation:
One of the most obvious pieces of misinformation . . . is that the 50 percent rise in paralytic poliomyelitis in 1958, and the real accelerated increase in 1959 have been caused by persons failing to be vaccinated This represents . . . an unwillingness to face facts and to evaluate the true effectiveness of the Salk vaccine. . . . A scientific examination of the data and the manner in which the data were manipulated, will reveal that the true effectiveness of the present Salk vaccine is unknown and greatly overrated.93
When pediatrician R. Mendelsohn, was asked whether polio would return if vaccinations were stopped, he replied "Doctors admit that forty percent of our population is not immunized against polio. So where is polio? Diseases are like fashions, they come and go . . ." Later on US National television he referred to epidemiological records which revealed the disappearance of polio in Europe during the 40's and 50's, without benefit of immunizations.94
Speaking at an international health convention in 1978, A. Burton reported that statistical data compiled by the University of New South Wales in Australia revealed that polio immunization programs had no measurable impact in reversing what was a recent epidemic in that country.
He expressed the view that polio comes in cycles anyway, and when it does subside, it is inadvertently considered "conquered" by vaccines.95 This naturally occurring cycle in polio epidemics was well illustrated in Great Britain where polio peaked in 1950, and had declined by 82 percent by the year 1956, at which time the vaccine was first introduced.96
Returning to the earlier cited US Congressional Hearings (HR 1054), we find that the nation of Israel experienced a major "type I" polio epidemic in 1958. Mass polio immunization had already been enforced and there was no appreciable difference in contraction levels between the vaccinated and unvaccinated.
Additionally, 3 years later in 1961, the state of Massachusetts experienced a "type II" polio outbreak in which "there were more paralytic cases in the triple vaccinates than in the unvaccinated".97
It is noteworthy that in one of the few double blind trials that have been conducted on a vaccine, was for the Salk polio vaccine, in which trial over 200 individuals who received the vaccine went on to contract polio, whereas no observed polio cases developed amongst the controls. This trial was reported by Mendelsohn who in the same 1984 article wrote:
The evidence points to mass inoculation against polio as the cause of most remaining cases of the disease . . . there is an ongoing debate among the immunologists regarding the . . . killed virus vs. live virus vaccine.
Supporters of the killed virus vaccine maintain that it is the presence of live virus organisms in the other product that is responsible for the polio cases that . . . appear.
Supporters of the live virus type argue that the killed virus vaccine offers inadequate protection and actually increases the susceptibility (to polio) of those vaccinated.
I believe that both factions are right, and that use of either of the vaccines will increase not diminish the possibility that your child will contract the disease.98
Thirteen scientists recently concluded that: vaccine failures in the major Oman polio epidemic could not be explained by failures in the cold chain, nor on suboptimum vaccine potency; the efficacy of OPV in inducing "humoral immunity" was lower than expected; and primary reliance on routine polio immunization may be "inadequate" to achieve the goal of eradicating polio by the year 2000. (They also noted similar paralytic polio epidemics in other highly vaccinated populations,99 e.g., the Gambia, Brazil, and Taiwan.)
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Data on Pertussis (Whooping Cough)
V. Fulginiti, Chairman of the American Academy of Paediatrics Committee on Infectious Diseases made this incisive observation:
Despite more than 30 years of experience with pertussis immunization, the reasons for recovery from the acute infection and subsequent immunity, are still uncertain. It is known that second attacks are rare following natural disease. It is also known that 45-95% of recipients of pertussis vaccine are susceptible to pertussis up to 12 years later . . . we do not understand the immunologic mechanisms involved in resistance to infection after natural disease or immunization.
Is pertussis vaccine effective? . . . prior to the widespread use ofpertussis vaccine, both the incidence of pertussis and the case-fatality ratio declined. A 50-fold reduction in incidence and an 84% reduction in case-fatality were recorded in Great Britain in the years between 1947 and 1972. . . . In England, protection provided by vaccines prior to 1968 was meager; no greater than 20% protection was noted. . . . Britain is in the position of advocating use of a vaccine for which there are not hard data.100
G.T. Stewart's observations as published in the British Medical Journal indicated that "of 8,092 cases of whooping cough, 2,940 (36%) were fully immunized, while only 2,424 (30%) were definitely not immunized."101
A Medical Tribune Report (January 10, 1979) details an outbreak of whooping cough in which 46 out of 85 fully immunized children contracted the disease.102 (the reason that the other 39 did not contract the disease could have been related to any number of predisposing factors).
Ekanem's earlier noted research (Table IX) , reveals an increase of 21 percent in the number whooping cough cases by the end of the three year period following implementation of an Expanded Program of Immunization in Nigeria.103
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Data on Tetanus Toxoid and Immune Globulin
Neustaedter indicates that "Tetanus seems to be nearly eliminated from the United States, primarily because of good hygiene and proper wound management." His research suggests that in the period of 1982-1984 in the US, there were a total of nine tetanus cases among both children and adolescents, in which there were no deaths.104
Whereas Coumoyer's research points to "contaminated umbilical stump infections" as a principal cause of tetanus in the Developing World.105
Such infections can be effectively rectified through providing appropriate information and training to traditional birth attendants. Both Cournoyer and Johnson indicate that there have been some reports of lock jaw death in properly inoculated individuals.106 & 107
Additionally Cournoyer suggests that "Evidence in support of the (tetanus toxoid) vaccine comes from epidemiologic studies which are by nature controversial, and which do not satisfy the criteria for scientific proof.108
According to the data contained in Table XVII, in the Dominican Republic the incidence of tetanus among children actually increased in the two year period following administration of tetanus toxoid.
Table XVIII indicates that in the same country, the rate of neonatal tetanus--among mothers underwent an increase in the year following administration of tetanus toxoid.109
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WHO SMALLPOX ERADICATION SUCCESS RECONSIDERED
Although smallpox is apparently now accorded to the history books, it will be necessary to re-examine the issue of this disease having been universally eradicated, with particular reference to the WHO eradication campaign. An honest look at this question is of considerable importance, as the current worldwide UCI-EPI program gains much of its legitimacy and inspiration from this widely acclaimed success story.
A strong challenge to this now popular view, is reflected in the post-campaign findings of medical researchers like Buttram and Hoffman:
Most people probably credit the smallpox vaccine with playing the major role in recent eradication of smallpox throughout the world, but let us examine the facts. In the article 'Vaccines a Future in Question,' statistics showed that less than 10 percent of children in developing countries have received vaccines.
They went on to comment that with this level of coverage, the WHO campaign was not a real factor in the eradication. Data obtained in their broad based research also led them to conclude that "mass smallpox vaccination was not necessary for the eradication of smallpox.110
In further examining this question from a longer historical perspective, it became readily apparent that the WHO claim did not at all square with the earlier data, i.e., historical smallpox eradication efforts. If we go back as far as the last century, we discover that Creighton's independent research findings as published in the Ninth Edition of the Encyclopedia Britannica, strongly contradict the effectiveness of mass smallpox immunization programs. A few revealing excerpts follow:
- . . . in Bavaria in 1871 of 30,742 cases 29,429 were in vaccinated persons, or 95.7 percent.
- Notwithstanding the fact that Prussia was the best re-vaccinated country in Europe, its mortality from smallpox in the epidemic of 1871 was higher (69,839) than any other Northern state.
- According to a competent statistician (A. Vogt), the death-rate from smallpox in the German army, in which all recruits are re-vaccinated, was 60 percent more than among the civil population of the same age . . . although re-vaccination is not obligatory among the latter.
- It is often alleged that the unvaccinated are so much inflammable material in the midst of the community, and that smallpox begins among them and gathers force so that it sweeps even the vaccinated before it. Inquiry into the facts has shown that at Cologne in 1870 the first unvaccinated person attacked by smallpox was the 174th in order of time, at Bonn the same year the 42nd, and at Liegnitz in 1871 the 225th.111
As we move on into the earlier part of this century we find the same dismal picture of increased susceptibility correlated with increased vaccination coverage. Dettman and Kalokerinos describe a visit they paid to the Philippines about 15 years ago:
. . . We were fortunate enough to address their own medical (and) health officials where we reminded them of the incidence of smallpox in formerly "immunized" Filipinos. We invited them to consult their own medical records and asked them to correct us if our own facts and figures disagreed.
No such correction has been forthcoming, and we can only conclude that between 1918-1919 there were 112,549 cases of smallpox notified, with 60,855 deaths. Systematic (mass) vaccination started in 1905, and since its introduction case mortality increased alarmingly. Their own records comment that "The mortality is hardly explainable." 112
Speaking at a 1973 environmental conference in Brussels, Professor George Dick admitted that in recent decades, 75 percent of those that have contracted smallpox in Britain, have had prior a history of vaccination. In that "only 40%" of children were vaccinated (and at most 10 percent of adults), such figures clearly indicate that the vaccinated--as in the much earlier historical record--continue to show a higher tendency to contract the disease.
Dick also admitted that smallpox had been eradicated in certain tropical countries without mass vaccination.113 (Table VIII reveals that in the 16 year period preceding the year the WHO eradication campaign was launched--38 additional countries had ceased to report any smallpox cases.)114
A. Hutchison writing in the Journal of the Royal Society in 1974, referred to the smallpox vaccines "lack of potency" and the inadequacies of other measures for containment, in his words, "I have given details of the various outbreaks of smallpox in Britain and where they were diagnosed. These clearly indicate that the (preventive) measures are most ineffective.115
An article in the New Scientist indicates that "The smallpox family of viruses is genetically unstable," and that new viral strains which threaten the "WHO smallpox eradication programme, could emerge anywhere.116 It is thus of interest that in a 1980 article in the Australasian Nurses Journal, Dettman and Kalokerinos pointed out that electron-microscopy cannot distinguish between the various "poxviruses.117 (According to D, de Saving of IDRC, as of 1990 DNA sequencing can make the distinquishingment. What is not known though, is whether this has any beating on the reporting of the various "pox" diseases worldwide.)
This fact led them to raise a vitally significant question "as to whether smallpox may be declared conquered, (it's estimated that only 10 percent of the world population actually received the vaccine) with the possibility of it masquerading under the guise of a similar pox." Their line of evidence and reasoning is summarily stated:
. . . we claim that if the evidence is honestly evaluated that smallpox has actually been prolonged and that the so called protective vaccinations actually put the recipient at risk from . . . the disease itself. Authorities now realize this and the 'top world' countries are making vociferous protests about third world countries continuing use of smallpox vaccination because:
(a) suddenly it has become recognized that it is an extremely dangerous procedure, (To give some idea of the vaccine's dangers, it was reported--in the late sixties--that annually, roughly 3,000 children were experiencing varying degrees of brain damage due to the smallpox vaccine;
and according to G. Kiftel in 1967, smallpox vaccination damaged the hearing of 3,296 children in West Germany, of which 71 became totally deaf.117) and
(b) it has now been conquered. The ultimate in ingenuity. . . .118
In turning to recognized textbooks on human virology and vertebrate viruses we find that attention has been given since 1970 to a disease called "monkeypox," which is said to be "clinically indistinguishable from smallpox." Cases of this disease have been found in Zaire, Cameroon, Nigeria, Ivory Coast, Liberia, and Sierra Leone (by May 1983, 101 cases have been reported).
It is observed that " . . . the existence of a virus that can cause clinical smallpox is disturbing, and the situation is being closely monitored."119 (For a highly detailed account of the history of this disease and efforts to eradicate it, which further corroborates these observations, see, Razzell P., The Conquest of Smallpox, Caliban Books, United Kingdom, 1977.) |