What is Acidosis?
The term acidosis is used to describe an increased acidity of the blood and other body tissues. Acidity is measured by the pH scale, which is a negative logarithmic scale of hydrogen ions. Thus, high acidity is represented by low pH values.
Acidosis describes an unhealthy concentration of hydrogen ions within your body fluids and tissues. Technically, the term acidemia is used to describe blood that is too acidic, whereas the term acidosis is used to describe the biochemical processes leading to over-acidity.
However, the two terms are often used interchangeably. Acidemia occurs when the blood falls below its optimal metabolic pH range of 7.35 to 7.45. Unbalanced metabolic activity, disrupted respiration, exposure to man-made and bio-toxins ... and consuming an imbalance of acidic foods are all primary contributors to the process of acidosis.
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Types of
Acidosis
The 3 common types of acidosis are Metabolic Acidosis, Lactic Acidosis and Respiratory Acidosis. Of these 3 types of acidosis ... Metabolic Acidosis is a primary concern and indicator when considering the factors in the degeneration of metabolic functions within the organism.
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Note - Acidosis if often misrepresented in mainstream North American medical, educational and scientific literature. Caution must be taken when reviewing such definitions and articles.
The folloiwng articles (below), clarify the significant and contrasting bio-chemistry differences between metabolic acidosis and lactic acidosis:
"The terms "lactate" or "lactic acid" need to be removed from any association with the cause of acidosis or the training that is used to delay the onset of acidosis." - Robert A. Robergs, et al.
- Exercise Physiology Laboratories, Department of Physical Performance and Development
Full Article: Acidosis - Biochemistry of Exercise-Induced Metabolic Acidosis
Synopsis: 80+ Years of Fundamental Bio-Chemistry Errors in Mainstream Medical Literature
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Metabolic Acidosis
Metabolic acidosis will be apparent in all degenerative disease conditions and disorders. Acidosis is one of the pre-conditions and primary indicators for degenerative disease and disorders in the organism.
ACIDOSIS, TOXICOSIS and HYPOXIA are the primary fundamental underlying cause of degenerative disease and disorders in society. Oxygen medicine research reverses these degerenative conditions and re-establish optimal metabolic conditions.
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Oxygen Medicine - Reversing Metabolic Acidosis
Oxygen medicine immediately begins reversing the underlying degenerative conditions of ACIDOSIS, TOXICOSIS and HYPOXIA ... bringing the body back to optimal metabolic conditions and function.
Oxygen medicine will safely flood the body with energized oxygen to eliminate the toxins in the body, raise oxygen levels, boost the immune system and re-balance the pH in the blood to optimal pH levels (pH 7.4).
Oxygen medicine nutrition will help to maintain optimal pH levels (pH 7.4) and optimal metabolic function for optimal quality of life.
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Metabolic Acidosis - Sugars and Candy
Most commercially made candies are made from high fructose corn syrup, processed sugar cane and/or artificial sweeteners such as aspartame and Splenda. These sugars and chemicals are quickly broken down by your digestive system and absorbed into your blood, where they affect its chemistry and pH levels (increase acid levels and lower pH levels). Insulin is needed to transport blood sugar into your cells.
Too many artificial sugars will cause TOXICOSIS in the organism. TOXICOSIS will impair the metabolism ... leading to ACIDOSIS and HYPOXIA. These primary fundamental underlying conditions will lead to innumerable degenerative conditions, including diabetes.
If you eat too much candy, or if your pancreas doesn’t produce enough insulin, or if your cells are desensitized to insulin, then too much blood glucose remains and potentially leads to health problems. Diabetics are often deterred from eating candy, although if they take too much insulin artificially, they may benefit from the readily available glucose that candy provides to bring their systems back into balance.
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Metabolic Acidosis - Medical Errors / Misinformation
The "Lactic Acid Concept" for causing Acidosis / Metabolic Acidosis:
"The lactic acidosis explanation of metabolic acidosis is not supported by fundamental biochemistry, has no research base of support, and remains a negative trait of all clinical, basic, and applied science fields and professions that still accept this construct.
The most important reason to discard the lactic acidosis concept is that it is invalid. It has no biochemical justification and, to no surprise, no research support. We have been criticized for our stance on the need to change how to teach and interpret metabolic acidosis."
- Robert A. Robergs, et al.
- Exercise Physiology Laboratories, Department of Physical Performance and Development
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Metabolic Acidosis - What are they trying to hide?
What is the cause of Acidosis? ... THE FACTS:
This is the crucial question that all physiologists must be able to answer. There are several examples of why the correct cause of metabolic acidosis needs to be accepted, communicated in education, and used in research interpretation and publication.
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Scientific validity.:
The most important reason to discard the lactic acidosis concept is that it is invalid. It has no biochemical justification and, to no surprise, no research support. We have been criticized for our stance on the need to change how to teach and interpret metabolic acidosis.
As such, the concept of a lactic acidosis remains evidence of 1920s academic and scientific inertia and a summary equation that does not represent cause and effect ... and out of simple convenience and apathy, still remains today.
We would hope that the academics and professionals from the basic and applied fields that continue to accept the lactic acidosis construct immediately change the way they teach and interpret this topic.
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Education:
Education is a powerful force that can induce change or reinforce error. Rather than continuing to reinforce error, educators need to recognize their power in reshaping how students and academics alike explain and discuss all matters pertaining to metabolic acidosis and skeletal muscle proton buffering. The correct teaching of metabolic acidosis is crucial for the promotion and acceptance of the correct understanding of exercise-induced metabolic acidosis.
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Sports Physiology, Coaching, and Training:
An acceptance of the true biochemistry of metabolic acidosis means that terms and descriptions used throughout sports physiology and coaching need to be changed. The terms "lactate" or "lactic acid" need to be removed from any association with the cause of acidosis or the training that is used to delay the onset of acidosis.
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Research of Strategies to Retard Exercise-Induced Metabolic Acidosis:
If it is assumed that lactate production causes acidosis, then it is a logical extension to hypothesize that reducing lactate production for a given cellular ATP demand should retard acidosis. If decreasing the rate of lactate production is accomplished by stimulating increased mitochondrial respiration [such as through dichloracetate infusion/ingestion (18)], such a strategy might also increase mitochondrial proton uptake and decrease/delay acidosis.
However, as is clear from the biochemistry, for a given rate of mitochondrial respiration, decreasing lactate production will decrease proton buffering and removal from skeletal muscle and increase the rate of onset and worsen the severity of acidosis.
On the basis of the biochemistry of muscle metabolism, the best way to decrease metabolic acidosis is to decrease nonmitochondrial ATP turnover by stimulating mitochondrial respiration. For a given ATP demand, any effort to decrease lactate production without increasing mitochondrial respiration will worsen metabolic acidosis.
- Robert A. Robergs et al. - Exercise Physiology Laboratories,
Department of Physical Performance and Development.
Full Article: Acidosis - Biochemistry of Exercise-Induced Metabolic Acidosis
Synopsis: 80+ Years of Fundamental Bio-Chemistry Errors in Mainstream Medical Literature
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